Heteromerization of Kir2.x potassium channels contributes to the phenotype of Andersen"s syndrome.

Regina Preisig-Müller; Günter Schlichthörl; Tobias Goerge; Steffen Heinen; Andrea Brüggemann; Sindhu Rajan; Christian Derst; Rüdiger W Veh; Jürgen Daut

doi:10.1073/pnas.102609499

Heteromerization of Kir2.x potassium channels contributes to the phenotype of Andersen"s syndrome.

Regina Preisig-Müller, Günter Schlichthörl, Tobias Goerge, Steffen Heinen, Andrea Brüggemann, Sindhu Rajan, Christian Derst (Co-author), Rüdiger W Veh, Jürgen Daut

Research output: Contribution to journal › Original Article › peer-review

165 Citations (Web of Science)

Original language	English
Pages (from-to)	7774-7779
Journal	PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA
Volume	99
Issue number	11
DOIs	https://doi.org/10.1073/pnas.102609499
Publication status	Published - 2002

Keywords

RECTIFYING K+ CHANNEL
FUNCTIONAL EXPRESSION
MOLECULAR-CLONING
DIFFERENTIAL DISTRIBUTION
MESSENGER-RNAS
HUMAN ATRIUM
RAT-BRAIN
PROTEINS
LOCALIZATION
HIPPOCAMPUS

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10.1073/pnas.102609499

Cite this

Preisig-Müller, R., Schlichthörl, G., Goerge, T., Heinen, S., Brüggemann, A., Rajan, S., Derst, C., Veh, R. W., & Daut, J. (2002). Heteromerization of Kir2.x potassium channels contributes to the phenotype of Andersen"s syndrome. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA , 99(11), 7774-7779. https://doi.org/10.1073/pnas.102609499

Preisig-Müller, Regina ; Schlichthörl, Günter ; Goerge, Tobias et al. / Heteromerization of Kir2.x potassium channels contributes to the phenotype of Andersen"s syndrome. In: PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA 2002 ; Vol. 99, No. 11. pp. 7774-7779.

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keywords = "RECTIFYING K+ CHANNEL, FUNCTIONAL EXPRESSION, MOLECULAR-CLONING, DIFFERENTIAL DISTRIBUTION, MESSENGER-RNAS, HUMAN ATRIUM, RAT-BRAIN, PROTEINS, LOCALIZATION, HIPPOCAMPUS",

author = "Regina Preisig-M{\"u}ller and G{\"u}nter Schlichth{\"o}rl and Tobias Goerge and Steffen Heinen and Andrea Br{\"u}ggemann and Sindhu Rajan and Christian Derst and Veh, {R{\"u}diger W} and J{\"u}rgen Daut",

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Preisig-Müller, R, Schlichthörl, G, Goerge, T, Heinen, S, Brüggemann, A, Rajan, S, Derst, C, Veh, RW & Daut, J 2002, 'Heteromerization of Kir2.x potassium channels contributes to the phenotype of Andersen"s syndrome.', PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA , vol. 99, no. 11, pp. 7774-7779. https://doi.org/10.1073/pnas.102609499

Heteromerization of Kir2.x potassium channels contributes to the phenotype of Andersen"s syndrome. / Preisig-Müller, Regina; Schlichthörl, Günter; Goerge, Tobias et al.
In: PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA , Vol. 99, No. 11, 2002, p. 7774-7779.

Research output: Contribution to journal › Original Article › peer-review

TY - JOUR

T1 - Heteromerization of Kir2.x potassium channels contributes to the phenotype of Andersen"s syndrome.

AU - Preisig-Müller, Regina

AU - Schlichthörl, Günter

AU - Goerge, Tobias

AU - Heinen, Steffen

AU - Brüggemann, Andrea

AU - Rajan, Sindhu

AU - Derst, Christian

AU - Veh, Rüdiger W

AU - Daut, Jürgen

PY - 2002

Y1 - 2002

KW - RECTIFYING K+ CHANNEL

KW - FUNCTIONAL EXPRESSION

KW - MOLECULAR-CLONING

KW - DIFFERENTIAL DISTRIBUTION

KW - MESSENGER-RNAS

KW - HUMAN ATRIUM

KW - RAT-BRAIN

KW - PROTEINS

KW - LOCALIZATION

KW - HIPPOCAMPUS

U2 - 10.1073/pnas.102609499

DO - 10.1073/pnas.102609499

M3 - Original Article

C2 - 12032359

SN - 0027-8424

VL - 99

SP - 7774

EP - 7779

JO - PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA

JF - PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA

IS - 11

ER -

Preisig-Müller R, Schlichthörl G, Goerge T, Heinen S, Brüggemann A, Rajan S et al. Heteromerization of Kir2.x potassium channels contributes to the phenotype of Andersen"s syndrome. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA . 2002;99(11):7774-7779. doi: 10.1073/pnas.102609499