Chlamydia pneumoniae infection acts as an endothelial stressor with the potential to initiate the earliest heat shock protein 60-dependent inflammatory stage of atherosclerosis.

Simone Kreutmayer; Adam Csordas; Jan Marco Kern; Viola Maass; Giovanni Almanzar; Martin Offterdinger; Robert Ollinger; Matthias Maass; Georg Wick

doi:10.1007/s12192-012-0378-7

Chlamydia pneumoniae infection acts as an endothelial stressor with the potential to initiate the earliest heat shock protein 60-dependent inflammatory stage of atherosclerosis.

Simone Kreutmayer, Adam Csordas, Jan Marco Kern (Co-author), Viola Maass, Giovanni Almanzar, Martin Offterdinger, Robert Ollinger, Matthias Maass, Georg Wick

Department of Labratory Medicine

Research output: Contribution to journal › Original Article (Journal) › peer-review

24 Citations (Web of Science)

Original language	English
Pages (from-to)	259-268
Journal	CELL STRESS & CHAPERONES
Volume	18
Issue number	3
DOIs	https://doi.org/10.1007/s12192-012-0378-7
Publication status	Published - 2013

Keywords

SERUM ANTIBODIES
CIGARETTE-SMOKE
CAROTID ATHEROSCLEROSIS
AUTOIMMUNE-DISEASE
ESCHERICHIA-COLI
T-CELLS
HEAT-SHOCK-PROTEIN-60
EXPRESSION
ASSOCIATION
MECHANISMS

Access to Document

10.1007/s12192-012-0378-7

Cite this

Kreutmayer, S., Csordas, A., Kern, J. M., Maass, V., Almanzar, G., Offterdinger, M., Ollinger, R., Maass, M., & Wick, G. (2013). Chlamydia pneumoniae infection acts as an endothelial stressor with the potential to initiate the earliest heat shock protein 60-dependent inflammatory stage of atherosclerosis. CELL STRESS & CHAPERONES, 18(3), 259-268. https://doi.org/10.1007/s12192-012-0378-7

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title = "Chlamydia pneumoniae infection acts as an endothelial stressor with the potential to initiate the earliest heat shock protein 60-dependent inflammatory stage of atherosclerosis.",

keywords = "SERUM ANTIBODIES, CIGARETTE-SMOKE, CAROTID ATHEROSCLEROSIS, AUTOIMMUNE-DISEASE, ESCHERICHIA-COLI, T-CELLS, HEAT-SHOCK-PROTEIN-60, EXPRESSION, ASSOCIATION, MECHANISMS",

author = "Simone Kreutmayer and Adam Csordas and Kern, {Jan Marco} and Viola Maass and Giovanni Almanzar and Martin Offterdinger and Robert Ollinger and Matthias Maass and Georg Wick",

year = "2013",

doi = "10.1007/s12192-012-0378-7",

language = "English",

volume = "18",

pages = "259--268",

journal = "CELL STRESS & CHAPERONES",

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Kreutmayer, S, Csordas, A, Kern, JM, Maass, V, Almanzar, G, Offterdinger, M, Ollinger, R, Maass, M & Wick, G 2013, 'Chlamydia pneumoniae infection acts as an endothelial stressor with the potential to initiate the earliest heat shock protein 60-dependent inflammatory stage of atherosclerosis.', CELL STRESS & CHAPERONES, vol. 18, no. 3, pp. 259-268. https://doi.org/10.1007/s12192-012-0378-7

Chlamydia pneumoniae infection acts as an endothelial stressor with the potential to initiate the earliest heat shock protein 60-dependent inflammatory stage of atherosclerosis. / Kreutmayer, Simone; Csordas, Adam; Kern, Jan Marco (Co-author) et al.
In: CELL STRESS & CHAPERONES, Vol. 18, No. 3, 2013, p. 259-268.

Research output: Contribution to journal › Original Article (Journal) › peer-review

TY - JOUR

T1 - Chlamydia pneumoniae infection acts as an endothelial stressor with the potential to initiate the earliest heat shock protein 60-dependent inflammatory stage of atherosclerosis.

AU - Kreutmayer, Simone

AU - Csordas, Adam

AU - Kern, Jan Marco

AU - Maass, Viola

AU - Almanzar, Giovanni

AU - Offterdinger, Martin

AU - Ollinger, Robert

AU - Maass, Matthias

AU - Wick, Georg

PY - 2013

Y1 - 2013

KW - SERUM ANTIBODIES

KW - CIGARETTE-SMOKE

KW - CAROTID ATHEROSCLEROSIS

KW - AUTOIMMUNE-DISEASE

KW - ESCHERICHIA-COLI

KW - T-CELLS

KW - HEAT-SHOCK-PROTEIN-60

KW - EXPRESSION

KW - ASSOCIATION

KW - MECHANISMS

U2 - 10.1007/s12192-012-0378-7

DO - 10.1007/s12192-012-0378-7

M3 - Original Article (Journal)

C2 - 23192457

SN - 1355-8145

VL - 18

SP - 259

EP - 268

JO - CELL STRESS & CHAPERONES

JF - CELL STRESS & CHAPERONES

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