Dysregulation of Copper Homeostasis: A Causative Mechanism in Non-alcoholic Fatty Liver disease?

Project: Research

PMU FFF E-09/10/053-PAT

Status	Finished
Effective start/end date	15/03/10 → 15/03/12

Glucose acts as a regulator of serum iron by increasing serum hepcidin concentrations.
Aigner, E. (First author), Felder, T. (Co-author), Oberkofler, H., Hahne, P., Auer, S. (Co-author), Soyal, S. M. (Co-author), Stadlmayr, A., Schwenoha, K., Pirich, C. (Co-author), Hengster, P., Datz, C. & Patsch, W. (Last author), 2013, In: JOURNAL OF NUTRITIONAL BIOCHEMISTRY . 24, 1, p. 112-117
Research output: Contribution to journal › Original Article › peer-review

55 Citations (Web of Science)
A role for low hepatic copper concentrations in nonalcoholic Fatty liver disease.
Aigner, E. (First author), Strasser, M. (Co-author), Haufe, H., Sonnweber, T., Hohla, F. (Co-author), Stadlmayr, A., Solioz, M., Tilg, H., Patsch, W. (Co-author), Weiss, G., Stickel, F. & Datz, C., 2010, In: AMERICAN JOURNAL OF GASTROENTEROLOGY . 105, 9, p. 1978-1985
Research output: Contribution to journal › Original Article › peer-review

162 Citations (Web of Science)

Project Details