Homeobox A9 transcriptionally regulates the EphB4 receptor to modulate endothelial cell migration and tube formation.

Thomas Bruhl; Carmen Urbich; Diana Aicher; Amparo Acker-Palmer; Andreas M Zeiher; Stefanie Dimmeler

doi:10.1161/01.RES.0000120861.27064.09

Homeobox A9 transcriptionally regulates the EphB4 receptor to modulate endothelial cell migration and tube formation.

Thomas Bruhl, Carmen Urbich, Diana Aicher, Amparo Acker-Palmer, Andreas M Zeiher, Stefanie Dimmeler

Universitätsklinik für Herzchirurgie

Publikation: Beitrag in Fachzeitschrift › Originalarbeit › Begutachtung

109 Quellenangaben (Web of Science)

Originalsprache	Englisch
Seiten (von - bis)	743-751
Fachzeitschrift	CIRCULATION RESEARCH
Jahrgang	94
Ausgabenummer	6
DOIs	https://doi.org/10.1161/01.RES.0000120861.27064.09
Publikationsstatus	Veröffentlicht - 2004

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10.1161/01.RES.0000120861.27064.09

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@article{2d71ddec462143fb95dd04dccd6d1b5f,

title = "Homeobox A9 transcriptionally regulates the EphB4 receptor to modulate endothelial cell migration and tube formation.",

keywords = "NITRIC-OXIDE SYNTHASE, ADHESION MOLECULE, TYROSINE KINASE, HOX GENES, ANGIOGENESIS, ACTIVATION, PHOSPHORYLATION, PROTEINS, BINDING, GROWTH",

author = "Thomas Bruhl and Carmen Urbich and Diana Aicher and Amparo Acker-Palmer and Zeiher, {Andreas M} and Stefanie Dimmeler",

year = "2004",

doi = "10.1161/01.RES.0000120861.27064.09",

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TY - JOUR

T1 - Homeobox A9 transcriptionally regulates the EphB4 receptor to modulate endothelial cell migration and tube formation.

AU - Bruhl, Thomas

AU - Urbich, Carmen

AU - Aicher, Diana

AU - Acker-Palmer, Amparo

AU - Zeiher, Andreas M

AU - Dimmeler, Stefanie

PY - 2004

Y1 - 2004

KW - NITRIC-OXIDE SYNTHASE

KW - ADHESION MOLECULE

KW - TYROSINE KINASE

KW - HOX GENES

KW - ANGIOGENESIS

KW - ACTIVATION

KW - PHOSPHORYLATION

KW - PROTEINS

KW - BINDING

KW - GROWTH

U2 - 10.1161/01.RES.0000120861.27064.09

DO - 10.1161/01.RES.0000120861.27064.09

M3 - Original Article

C2 - 14764452

SN - 0009-7330

VL - 94

SP - 743

EP - 751

JO - CIRCULATION RESEARCH

JF - CIRCULATION RESEARCH

IS - 6

ER -