Dysregulation of Copper Homeostasis: A Causative Mechanism in Non-alcoholic Fatty Liver disease?

Projekt: Forschung

PMU FFF E-09/10/053-PAT

Status	Abgeschlossen
Tatsächlicher Beginn/ -es Ende	15/03/10 → 15/03/12

Glucose acts as a regulator of serum iron by increasing serum hepcidin concentrations.
Aigner, E. (Erstautor/-in), Felder, T. (Co-Autor/-in), Oberkofler, H., Hahne, P., Auer, S. (Co-Autor/-in), Soyal, S. M. (Co-Autor/-in), Stadlmayr, A., Schwenoha, K., Pirich, C. (Co-Autor/-in), Hengster, P., Datz, C. (Co-Autor/-in) & Patsch, W. (Letztautor/-in), 2013, in: JOURNAL OF NUTRITIONAL BIOCHEMISTRY . 24, 1, S. 112-117
Publikation: Beitrag in Fachzeitschrift › Originalarbeit › Begutachtung

58 Quellenangaben (Web of Science)
A role for low hepatic copper concentrations in nonalcoholic Fatty liver disease.
Aigner, E. (Erstautor/-in), Strasser, M. (Co-Autor/-in), Haufe, H., Sonnweber, T., Hohla, F. (Co-Autor/-in), Stadlmayr, A., Solioz, M., Tilg, H., Patsch, W. (Co-Autor/-in), Weiss, G., Stickel, F. & Datz, C. (Letztautor/-in), 2010, in: AMERICAN JOURNAL OF GASTROENTEROLOGY . 105, 9, S. 1978-1985
Publikation: Beitrag in Fachzeitschrift › Originalarbeit › Begutachtung

177 Quellenangaben (Web of Science)

Projektdetails